Jamun herb Eugenia jambolana plant medicinal properties
Jamun seed has gastro-protective properties mainly through promotion of mucosal defensive factors and antioxidant status and decreasing lipid peroxidation. Jamun also has anti cancer and anti viral potential.
Jamun fruit extract and breast cancer
Eugenia jambolana Lam. berry extract inhibits growth and induces apoptosis of
human breast cancer but not non-tumorigenic breast cells.
J Agric Food Chem. 2009 February. Li L, Adams LS, Chen S, Killian C, Ahmed A,
Seeram NP. Department of Biomedical and Pharmaceutical Sciences, Bioactive
Botanical Research Laboratory, College of Pharmacy, University of Rhode Island,
Kingston, Rhode Island, USA.
The ripe purple berries of the native Indian plant Eugenia jambolana Lam., known
as Jamun, are popularly consumed and available in the United States in Florida
and Hawaii. Despite the growing body of data on the chemopreventive potential of
edible berry extracts, there is paucity of such data for Jamun fruit. Therefore
our laboratory initiated the current study with the following objectives: to
prepare a standardized Jamun fruit extract for biological studies and to
investigate the antiproliferative and pro-apoptotic effects of Jamun fruit
extract in estrogen dependent/aromatase positive (MCF-7aro), and estrogen
independent (MDA-MB-231) breast cancer cells, and in a normal/nontumorigenic
(MCF-10A) breast cell line. Jamun fruit extract was standardized to anthocyanin
content 3.5% anthocyanins (as cyanidin-3-glucoside equivalents) which occur as
diglucosides of five anthocyanidins / aglycons: delphinidin, cyanidin, petunidin,
peonidin and malvidin. In the proliferation assay, Our studies suggest that
Jamun fruit extract may have potential beneficial effects against breast cancer.
Jamun has antiviral activity
In vitro antiviral activity of plant extracts on goatpox virus replication.
Indian J Exp Biol. 2008 Feb;
Bhanuprakash V, Hosamani M, Balamurugan V, Gandhale P, Naresh R, Swarup D, Singh
RK. Division of Virology, Indian Veterinary Research Institute, Mukteswar 263
138, India.
Four plants having known medicinal properties were screened for inhibition of
goatpox virus (GTPV) replication in vitro. Of the 4 plants, extract of Acacia
arabica (Babul) and Eugenia jambolana ( Jamun ) leaves had inhibition (%) 99.70
and 99.92 at their maximum non toxic concentrations, 99.93 +/- 0.38 and 1999.73
+/- 0.50 microg/ml, respectively in all cytopathic effect (CPE) inhibition
assays. Results indicated that the extract of Acacia arabica and Jamun leaves
inhibited GTPV replication in vitro.
Liver health
Hepatoprotective activity of Eugenia jambolana Lam. in carbon tetrachloride
treated rats.
Indian J Pharmacol. 2009 Feb; Sisodia SS, Bhatnagar M. Department of
Pharmacology, Bhupal Nobles' Girls College of Pharmacy, Udaipur, Rajasthan,
India.
To estimate the hepatoprotective effects of the methanolic seed extract of
Eugenia jambolana Lam. (Myrtaceae), in Wistar albino rats treated with carbon
tetrachloride (CCl(4)). Liver damage in rats treated with CCl(4) administered
subcutaneously, on alternate days for one week) was studied by assessing
parameters such as serum glutamate oxaloacetate transaminase (SGOT), serum
glutamate pyruvate transaminase (SGPT), alkaline phosphatase (ALP), acid
phosphatase (ACP) and bilirubin (total and direct). The effect of
co-administration of Eugenia jambolana Lam on the above parameters was
investigated. These biochemical observations were supplemented by weight and
histological examination of liver sections. Liv.52((R)) was used as positive
control. Data were analyzed by one way ANOVA, followed by Scheff's/Dunnett's
test. Administration of Eugenia jambolana Lam. significantly prevented carbon
tetrachloride induced elevation of serum SGOT, SGPT, ALP, ACP and bilirubin
(total and direct) level. Histological examination of the liver section revealed
hepatic regeneration, after administration of various doses of Eugenia jambolana
Lam. The results were comparable to that of Liv.52). The study suggests
preventive action of Jamun in carbon tetrachloride induced liver toxicity.
Hepatic cell regeneration process was dose dependent.
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